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As a commonly used clinic chemotherapeutic agent, cisplatin resistance in cancer is associated with the decreased intracellular drug accumulation, and the enhanced DNA repair capacity along with dysregulation of ferroptosis-related signaling pathways

Studies show that the XPC variants are associated with increased DNA adduct levels after aflatoxin B1 exposure, increasing liver cancer risk.[ref][ref][ref] This means that for people who both accumulate more DNA damage from mycotoxins (because of detox variants) and repair it less efficiently (because of XPC variants), the longterm cancer risk from chronic exposure is significantly higher

A new study shows that yeast exposed to cadmium cleverly reprogram the transcriptome and proteome to protect themselves from its toxic effects

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