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It accomplishes this through several distinct mechanisms: Collagen and elastin stimulation: GHK-Cu upregulates the genes responsible for collagen I, collagen III, and elastin synthesis the structural proteins that give skin its firmness, bounce, and thickness

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This exacerbates lipid peroxidation, drives excessive ROS accumulation and neuroinflammation, and ultimately triggers ferroptosis, resulting in irreversible neuronal damage and neurotoxicity (Chen et al., 2025a)