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Future studies will reveal the applicability of our model, for example, to activate enzymatically inactive glutaredoxins, to alter the sensitivity and responsiveness of glutaredoxin-coupled fluorescent redox sensors, to further address the catalytic mechanism with GSSCys, HEDS and physiological substrates, or to design specific transition-state inhibitors

Toxicol Appl Pharmacol 2011;251:22633 [DOI] [PMC free article] [PubMed] [Google Scholar] 25.Yan M, Huo Y, Yin S, Hu H.Mechanisms of acetaminophen-induced liver injury and its implications for therapeutic interventions

In mouse model, Frakes et al

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