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For instance, hypoxia can drive the transformation of macrophages into the M1 pro-inflammatory phenotype by stabilizing hypoxia-inducible factor-1 (HIF-1), promote glycolytic reprogramming of activated naive B cells and naive T cells, and simultaneously inhibit the immunosuppressive function of regulatory T cells (Tregs) thereby triggering and amplifying inflammatory responses

[DOI] [PMC free article] [PubMed] [Google Scholar] 41.Patenaude A, Murthy MRV, Mirault M-E: Mitochondrial Thioredoxin System: EFFECTS OF TrxR2 OVEREXPRESSION ON REDOX BALANCE, CELL GROWTH, AND APOPTOSIS

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