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The underlying mechanism involves AS-IVs ability to reduce mitochondrial ROS in myocardial cells of MI mice, inhibit NLRP3 inflammasome activation, and subsequently downregulate the expression of downstream inflammatory factors such as IL-18 and IL-1, along with the caspase-1 regulator involved in cell pyroptosis

But with that power comes risk

Its extended in vitro stability profile makes it suitable for receptor pharmacology research requiring sustained agonist activity across long incubation windows

Figure 5: The average number of crossing into the target quadrant in each of the four different experimental groups included in the first part of the study