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This study will greatly contribute to the the current literature in these respects

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Secondary coenzyme Q 10 deficiency results from mutations or deletions in genes that are not directly related to coenzyme Q 10 biosynthetic pathway

Intracellular glutathione also followed the trend of GCLC, exhibiting an NFT-dependent increase that was reversed by anti Nrf2-siRNA to levels comparable to those observed in untreated HepG2 cells (Fig