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13 This provides the basis for augmenting GSH concentrations against a wide variety of pathophysiologic states, including hepatic dysfunction or cirrhosis, or conditions affecting the epithelial cells, which can use exogenous GSH for protection

In mesothelioma models, SFN disrupts PRMT5/MEP50 complex function, inhibiting its methyltransferase activity, reactivating tumor suppressor expression, and suppressing cancer cell proliferation, invasion, and stem-like properties (79, 80)

At the core of breast carcinogenesis is epigenetic disruption, which involves changes in DNA methylation, histone modifications, and non-coding RNA

"N-acetylcysteine replenishes glutathione in HIV infection." Eur J Clin Invest