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Exercise, as a critical adjunct to pharmacological therapy, has been extensively shown to confer multiple therapeutic benefits in NDDs, including suppression of neuronal ferroptosis, mitigation of neuroinflammation, reduction of Tau hyperphosphorylation, and enhancement of cognitive performance (Gutierre et al., 2025)

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At the body level, kidneys, lungs, and intestines consume most of the circulating GSH [15], highlighting the important oxidative stress that might occur in these organs [1]