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Trends with exposure for all neoplastic lung lesions were highly significant for female rats

Inhalation of mainstream cigarette smoke or the vapor phase smoke component, 1,3-butadiene, was shown to promote plaque development in cockerels, but CO or an injection of NNK or solubilized concentrated cigarette smoke condensate from an unventilated 2R1 reference cigarette did not promote plaque development in cockerels

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