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In 2012, Stanford neuroimmunologist Larry Steinman, MD (whose groundbreaking multiple-sclerosis research led directly to the development and, in 2004, FDA approval of the widely used MS drug Tysabri), along with Jonathan Rothbard, PhD, a senior scientist in Steinman's lab, and their colleagues found, surprisingly, that a sawed-off little protein by the name of beta-amyloid possibly the most reviled molecule in the history of medicine because its build-up in the brain is frequently (although perhaps wrongly) deemed the villain in Alzheimer's disease had a therapeutic effect in a rodent model of MS

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