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To evaluate potential effect modification, we performed subgroup analysis, stratified by allergic sensitization, age and gender, separately

40 In COPD animal models, CS disrupts mitochondrial structure in ASMCs and activates the phosphatidylinositol 3-kinase (PI3K)/protein kinase B (AKT)/mechanistic target of rapamycin (mTOR) signaling pathway, and leading to oxidative stress and metabolic dysfunction