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Histaminylation of glutamine residues is a novel posttranslational modification implicated in G-protein signaling

After coronary artery ligation in mice, intraperitoneal T4 treatment upregulated cardiac ILK and Akt activity, enhanced early myocyte survival, and improved cardiac function establishing the mechanistic basis for the broader tissue-repair claims associated with thymosin beta-4 and its 1723 fragment TB-500

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